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For years bodybuilders have experimented with various compounds while in their cutting phases to find the ultimate AAS stack to assist in cutting body fat while preserving lean body massfor an optimal build. There are, however, some common myths and misconceptions about AAS, which are discussed in this article. If you are not familiar with these myths (and it is very easy to find AAS-related news in almost every social media platform; check out the Internet at your own risk), then be sure to refer to the article by Dr, craft ultimate stacker storage. Steve Phinney for a few of the more common misconceptions, craft ultimate stacker storage. Myth #1: Low and Slow Dieting Causes Loss of Muscle Mass Failed experiments have demonstrated that when the body weight is cut to 20% of baseline after 2 weeks of low- and slow-dieting, the muscle mass has actually increased, not the lean mass (which is an important distinction). And more importantly, the gains in lean mass were also very significant, as measured by muscle volume in a laboratory setting; the researchers measured both maximal and submaximal strength gains, and the gains were over and above those seen with low- and slow dieting.  This was not due to the addition of calories; rather, the increase in muscle volume was due to the fact that the body is getting a large dose of oxygen (anaerobic exercise) as compared to the normal level of carbon dioxide; consequently the body has a greater capacity to regenerate the muscle cells and tissues and therefore, the overall muscle mass, deca durabolin what does it do. Myth #2: AAS Disrupts Muscle Fiber Function Several research studies have demonstrated that both acute AAS administration and chronic low- and slow-dieting do not significantly modify the fiber type composition or the function of muscle fibers. Specifically, a study of rats in the laboratory has demonstrated that chronic low- and slow-dieting do not alter the ratio of fast-twitch to slow-twitch fiber type or the expression level of the muscle fiber type in vivo. In addition, a study has demonstrated that acute AAS administration does not impair the ability of fast-twitch fibers to provide energy by generating additional ATP, whereas slow-twitch fibers continue to generate ATP during endurance exercise; furthermore, the expression of several mitochondrial proteins has not been demonstrated to be altered to any appreciable degree by chronic AAS, ultimate stacker craft storage. [2, 3, 4] And finally, the study investigating the effects of AAS administration on the muscle protein synthetic rate in vivo has shown that only brief AAS treatment (0.1-0.5 mg/kg/day) does not significantly affect the rate of muscle protein synthesis but does enhance the rate of
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